Noninvasive diagnosis of ruptured peripheral atherosclerotic lesions and myocardial infarction by antibody profiling
J. Clin. Invest. Kitty B.J.M. Cleutjens, et al. 118:2979 doi:10.1172/JCI32767 [
Go to this article.]

Figure 1Autoantibody signature of 4 phage-displayed peptides in pooled sera of patients with proven peripheral ruptured (black bars) or stable (white bars) lesions and in pooled control sera (gray bars). Reactivity is represented as the ratio of OD
450 sample/(mean OD
450 + 3SD) for empty phage. Clones A1, A7, and E1 had identical inserts. Data are mean ± SD. *
P < 0.05.